Glycemic variability correlates strongly with postprandial β-cell dysfunction in a segment of type 2 diabetic patients using oral hypoglycemic agents.
Kohnert KD, Augstein P, Zander E et al.
Editor’s note: The fetal insulin hypothesis (Lancet 1999;353:1789–92) proposes that the association between low birth weight (LBW) and later development of type 2 diabetes is a result of genetic variations that predispose to both characteristics, possibly as a result of reduced insulin secretion rather than the original suggestion of fetal malnutrition.
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